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collagen type iii level  (Cusabio)


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    Cusabio collagen type iii level
    In the in vitro model of arachnoid cell fibrosis, the expression of miR-30a decreased, and the expression of TRAF3IP2 was upregulated. (a) Differential mRNA expression of miR-30a, TRAF3IP2, and TGF-β1 in arachnoid cells between the fibrotic group and control group (** P < 0.01 compared with the control group). (b) Differential protein expression of TRAF3IP2, TGF-β1, and α-SMA in arachnoid cells between the fibrotic and control groups (** P < 0.01 compared with the control group; * P < 0.05 compared with the control group). (c) Differential content of collagen I and <t>collagen</t> <t>III</t> in the model of arachnoid cell fibrosis (** P < 0.01 compared with the control group).
    Collagen Type Iii Level, supplied by Cusabio, used in various techniques. Bioz Stars score: 92/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/collagen type iii level/product/Cusabio
    Average 92 stars, based on 4 article reviews
    collagen type iii level - by Bioz Stars, 2026-03
    92/100 stars

    Images

    1) Product Images from "Decreased MiR-30a promotes TGF-β1-mediated arachnoid fibrosis in post-hemorrhagic hydrocephalus"

    Article Title: Decreased MiR-30a promotes TGF-β1-mediated arachnoid fibrosis in post-hemorrhagic hydrocephalus

    Journal: Translational Neuroscience

    doi: 10.1515/tnsci-2020-0010

    In the in vitro model of arachnoid cell fibrosis, the expression of miR-30a decreased, and the expression of TRAF3IP2 was upregulated. (a) Differential mRNA expression of miR-30a, TRAF3IP2, and TGF-β1 in arachnoid cells between the fibrotic group and control group (** P < 0.01 compared with the control group). (b) Differential protein expression of TRAF3IP2, TGF-β1, and α-SMA in arachnoid cells between the fibrotic and control groups (** P < 0.01 compared with the control group; * P < 0.05 compared with the control group). (c) Differential content of collagen I and collagen III in the model of arachnoid cell fibrosis (** P < 0.01 compared with the control group).
    Figure Legend Snippet: In the in vitro model of arachnoid cell fibrosis, the expression of miR-30a decreased, and the expression of TRAF3IP2 was upregulated. (a) Differential mRNA expression of miR-30a, TRAF3IP2, and TGF-β1 in arachnoid cells between the fibrotic group and control group (** P < 0.01 compared with the control group). (b) Differential protein expression of TRAF3IP2, TGF-β1, and α-SMA in arachnoid cells between the fibrotic and control groups (** P < 0.01 compared with the control group; * P < 0.05 compared with the control group). (c) Differential content of collagen I and collagen III in the model of arachnoid cell fibrosis (** P < 0.01 compared with the control group).

    Techniques Used: In Vitro, Expressing, Control

    MiR-30a inhibits subarachnoid fibrosis through inhibiting TRAF3IP2. (a and b) The mRNA and protein expression of miR-30a, TRAF3IP2, TGF-β1 and α-SMA from different groups (** P < 0.01 compared with the control group; *** P < 0.001 compared with the control group). (c) The content of collagen I and collagen III from different groups (*** P < 0.001 compared with the control group. ** P < 0.01 compared with the control group; * P < 0.05 compared with the control group).
    Figure Legend Snippet: MiR-30a inhibits subarachnoid fibrosis through inhibiting TRAF3IP2. (a and b) The mRNA and protein expression of miR-30a, TRAF3IP2, TGF-β1 and α-SMA from different groups (** P < 0.01 compared with the control group; *** P < 0.001 compared with the control group). (c) The content of collagen I and collagen III from different groups (*** P < 0.001 compared with the control group. ** P < 0.01 compared with the control group; * P < 0.05 compared with the control group).

    Techniques Used: Expressing, Control



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    collagen type iii level  (Cusabio)
    92
    Cusabio collagen type iii level
    In the in vitro model of arachnoid cell fibrosis, the expression of miR-30a decreased, and the expression of TRAF3IP2 was upregulated. (a) Differential mRNA expression of miR-30a, TRAF3IP2, and TGF-β1 in arachnoid cells between the fibrotic group and control group (** P < 0.01 compared with the control group). (b) Differential protein expression of TRAF3IP2, TGF-β1, and α-SMA in arachnoid cells between the fibrotic and control groups (** P < 0.01 compared with the control group; * P < 0.05 compared with the control group). (c) Differential content of collagen I and <t>collagen</t> <t>III</t> in the model of arachnoid cell fibrosis (** P < 0.01 compared with the control group).
    Collagen Type Iii Level, supplied by Cusabio, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/collagen type iii level/product/Cusabio
    Average 92 stars, based on 1 article reviews
    collagen type iii level - by Bioz Stars, 2026-03
    92/100 stars
    		  Buy from Supplier

    Image Search Results


    In the in vitro model of arachnoid cell fibrosis, the expression of miR-30a decreased, and the expression of TRAF3IP2 was upregulated. (a) Differential mRNA expression of miR-30a, TRAF3IP2, and TGF-β1 in arachnoid cells between the fibrotic group and control group (** P < 0.01 compared with the control group). (b) Differential protein expression of TRAF3IP2, TGF-β1, and α-SMA in arachnoid cells between the fibrotic and control groups (** P < 0.01 compared with the control group; * P < 0.05 compared with the control group). (c) Differential content of collagen I and collagen III in the model of arachnoid cell fibrosis (** P < 0.01 compared with the control group).

    Journal: Translational Neuroscience

    Article Title: Decreased MiR-30a promotes TGF-β1-mediated arachnoid fibrosis in post-hemorrhagic hydrocephalus

    doi: 10.1515/tnsci-2020-0010

    Figure Lengend Snippet: In the in vitro model of arachnoid cell fibrosis, the expression of miR-30a decreased, and the expression of TRAF3IP2 was upregulated. (a) Differential mRNA expression of miR-30a, TRAF3IP2, and TGF-β1 in arachnoid cells between the fibrotic group and control group (** P < 0.01 compared with the control group). (b) Differential protein expression of TRAF3IP2, TGF-β1, and α-SMA in arachnoid cells between the fibrotic and control groups (** P < 0.01 compared with the control group; * P < 0.05 compared with the control group). (c) Differential content of collagen I and collagen III in the model of arachnoid cell fibrosis (** P < 0.01 compared with the control group).

    Article Snippet: ELISA kit was used to detect the TGF-β1 level (EK0514, Wuhan Boster Biological Technology, Ltd, China), collagen type I level (CSB-E08084r, CUSABIO, China), and collagen type III level (CSB-E07924r, CUSABIO, China) following the protocols constructions.

    Techniques: In Vitro, Expressing, Control

    MiR-30a inhibits subarachnoid fibrosis through inhibiting TRAF3IP2. (a and b) The mRNA and protein expression of miR-30a, TRAF3IP2, TGF-β1 and α-SMA from different groups (** P < 0.01 compared with the control group; *** P < 0.001 compared with the control group). (c) The content of collagen I and collagen III from different groups (*** P < 0.001 compared with the control group. ** P < 0.01 compared with the control group; * P < 0.05 compared with the control group).

    Journal: Translational Neuroscience

    Article Title: Decreased MiR-30a promotes TGF-β1-mediated arachnoid fibrosis in post-hemorrhagic hydrocephalus

    doi: 10.1515/tnsci-2020-0010

    Figure Lengend Snippet: MiR-30a inhibits subarachnoid fibrosis through inhibiting TRAF3IP2. (a and b) The mRNA and protein expression of miR-30a, TRAF3IP2, TGF-β1 and α-SMA from different groups (** P < 0.01 compared with the control group; *** P < 0.001 compared with the control group). (c) The content of collagen I and collagen III from different groups (*** P < 0.001 compared with the control group. ** P < 0.01 compared with the control group; * P < 0.05 compared with the control group).

    Article Snippet: ELISA kit was used to detect the TGF-β1 level (EK0514, Wuhan Boster Biological Technology, Ltd, China), collagen type I level (CSB-E08084r, CUSABIO, China), and collagen type III level (CSB-E07924r, CUSABIO, China) following the protocols constructions.

    Techniques: Expressing, Control